Could be Parkinson's a lack of mainly B vitamins?

IS PARKINSON'S A MULTI-CARENTIAL SYNDROME IN WHICH THE B-GROUP VITAMINS PLAY AN ESSENTIAL ROLE?

"The truth is the truth, whether it's said

by (the king) Agamemnon or his servant."

Antonio Machado

Could be what we call Parkinson's a lack of mainly B vitamins?

Could a good part of what we know as Parkinson's be a syndrome of multiple deficiencies (especially, vitamin C and glutathione, as well as magnesium, zinc and all the B-group vitamins?

If we look at the functions of each B vitamin and the symptoms that lead to its deficiency - which are all of the symptoms of Parkinson's - suspicion begins to arise. And considering that Levodopa-Carbidopa affects vitamins B3, B6 and B9 at least, it increases suspicion.

Let's focus on the B vitamins. These studies show the highest risk of Parkinson's when there is a deficit (a Dutch study, led by De Lau, states that a low level of vitamin B6 -less than 0.25 mg per day, when the recommended daily amount would be around 1 or 2 mg per day- increases the risk of Parkinson's by 54 %) and the lowest risk when there is the highest intake within the recommended range (up to 51 % with vitamin B2 or 49 % with B9).

PARKINSON'S DISEASE AND THE IMPORTANCE OF EACH AND EVERY B VITAMINS TO PREVENT, SLOW DOWN AND IMPROVE SYMPTOMS.

  Studies showing the benefits of each vitamin for Parkinson's:

  Vitamin B1 -

  Food (Iber 1982, Mousseau 1996);

  Supplements (Luong and Nguyen 2012; Costantini 2016)

  Vitamin B2 -

  Food (McCormick 1988 - 51 % lower risk -; Powers 1999)

  Supplements (Coimbra 2003 -symptom improvement 44 to 77 %-,      Jornayvaz 2010, Marashly 2017).

  Vitamin B3 -

  Food (Fall 1999, Hellenbrand 1996, Vrecko 1993);

  Supplements (Kuhn 1993, Black 1986)

  Vitamin B6 -

  Food (Frankl 1977, Lau 2006 -lower level, 54 % more risk, Murakami 2010);

  Supplements (Baker 1941, Lewis 2002, Vainstok 1979, Tan 2005)

  Vitamin B9 -

  Food (Lau 2006);

  Supplements (Duan 2002, Mattson 2003, Postuma 2006, Schwartz 1992,

  Religa 2006 - 49 % less risk).

  Vitamin B12 -

  Food (Lau 2006, Quershi 2011);

  Supplements (Rajabally 2013, Christine 2018, McCarter 2019,

  Schaffner 2019 - a major improvement in mobility).

                       (Jesus Marquez Rivera. Parkinson here and now).

Could excessive oxidation, excess iron or aluminum, neuroinflammation, malfunctioning of mitochondria, problems with alpha-synuclein, the inability to "correct" the genes that predispose to Parkinson's with the nutrients with greater epigenetic potential (vitamin D, B12, magnesium, vitamin C ...) be normal with such deficiencies?

Marashly says verbatim in her 2017 study:

"Riboflavin protects against neurotoxicity by improving oxidative stress, mitochondrial dysfunction, neurogenic inflammation, glutamate excitotoxicity and homocysteine neurotoxicity. Oxidative stress, mitochondrial dysfunction, neurogenic inflammation, glutamate excitotoxicity and homocysteine neurotoxicity are all implicated in neurodegeneration and neurotoxicity."

Vitamin C activates folic acid and recycles oxidized glutathione, vitamin B2 is needed to activate vitamin B6 (WITHOUT WHICH DOPA CANNOT BE CONVERTED INTO DOPAMINE) and produce glutathione.

Another piece of the puzzle…